Three Different MM types:

Skeletal

Cardiac

Smooth

AKA's: Stiated, Voluntary

attached to skeleton

Stiated- appeared striped

Voluntary- can control their movement

SEVERAL SHEATHED M FIBERS ARE BUNDLED TOGETHER AND WRAPPED BY THIS

ALL THESE BUNDLES FORM THE FASCICLE

COVERS ENTIRE LENGTH OF MM

A BUNCH OF FASCICLES WRAPPED TOGETHER

TAPERS INTO TENDON THAT ATTACHES TO BONE

NONSTIATED

INVOLUNTARY

FOUND IN HOLLOW TUBESOF VISCERAL ORGANS (STOMACH, URINARY BLADDER, RESPIRATORY PASSAGE)

PROPELS FOOD AND SUBSTANCES THROUGH PERISTOLISIS

2 LAYERS:

CIRCULAR LAYER

LONGITUDAL LAYER

THIS ARRANGEMENT ALLOWS MM ALTERNATELY CONTRACT AND RELAX

PRODUCE MOVEMENT

MAINTAINS POSTURE

STABILIZES JOINTS (BY PRODUCT IS ATP)

GENERATES HEAT

LUMBER EXTENSORS AE LIKELY TO BE DEFICIENT BC IF YOU DONT USE IT YOU LOSE IT AND IT TURNS TO FAT

WEAK MM, POOR STABILIZERS INADEQUATE PROTECTION

BECOMES INFLAMMED TO PROTECT

USE STRENGTH TRAINING, NEUROLOGICAL TRAINING, AND ADJUSTMENT TO HELP

MYOFILBRILS ARE CHAINS OF THESE

LINED END TO END LIKE COKE CANS

AREA BTW TWO Z LINES

ACTIN (THIN FILAMENTS)- CONTRACTILE PROTEIN, ALSO PREVENTS CONTRACTION 

MYOSIN (THICK FILAMENT/PROTEIN)- CONTAIN ATPase WHICH SPLIT ATP TO GENERATE FOR CONTRACTION

SURROUNDS EVERY MYOFIBRIL

STORES AND RELEASE CALCIUM WHEN MM FIBER IS STIMULATED OR CONTRACTED

IN ORDER FOR MM TO CONTRACT:

-IRRITABILITY- ABILITY TO RECIEVE AND RESPOND

-CONTRACTILITY- THE ABILITY TO SHORTEN WHEN STIMULUS IS RECIEVED

-SKELETAL MM MUST BE STIMULATED BY NERVE IMPULSE TO CONTRACT (ACTION POTENTIAL)

-FORM A MOTOR UNIT OF MM TISSUES AND MOTOR NERVES

MM CELLS REACT TO STIMULI WITH THIS

"ALL OF NONE" NO PARTIAL CONTRACTION OCCURS

BEING ABLE TO VARY THE RESPONSE OF A MM CONTRACTION

OCCURS IN 2 WAYS:

BY CHANGING FREQUENCY OF STIMULI (SPEED)

CHANGING THE STRENGTH OF STIMULI (WEIGHT)

NERVE IMPULSES ARE SUMMED AND CONTRACTION OF MM GETS STRONGER AND SMOOTHER

*WHEN YOU PICK UP HEAVIER THINGS YOU RECRIUT MORE TETANUS

TAKE ATP... 3 PATHWAYS TO GENERATE ATP:

1. DIRECT PHOSPHORYLATION OF ADP BY CREATINE PHOSPHATE

2. AEROBIC RESPIRATION (OXIDATIVE PHOS. 36 ATP)

3. ANAEROBIC GLYCOLYSIS AND LACTIC ACID FORMATION (2 ATP FASTER THAN AR THOUGH)

*ATP LAST ABOUT 4-6 SECONDS

WHEN THE MM SHORTENS

EX. BENDING THE KNEE

WHEN THE MM LENGTHENS

EX. TRYING TO PUSH A WALL

SKELETAL MM ACTIVITY IS CONSTANTLY OCCURING

ALLOWS TO STAY FIRM/HEALTHY

CALLED "MM TONE"

NO STIMULATION--> ATROPHY

STIMULATION--> HYPERTROPHY

STRONGER MORE FLEXIBLE MM... BC BLOOD SUPPLY INCREASES MORE MM CELLS FORM, MORE MITOCHONDRIA, MORE OXYGEN STORED.