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CVA = "The ______ onset of neurological dysfxn resulting from an ___________ of _______ blood flow that lasts for ________ ." |
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acute, abnormality, cerebral |
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Ischemic CVA usually 2º to: (3) |
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thrombus, embolus, conditions >> low perfusion pressures (e.g. MI) |
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CVA: ___(#)___ leading cause of death of adults in US ___(#)___ leading cause of disability of adults in US |
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Which gender experiences more CVAs? |
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Which race more common for CVA? |
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% of CVAs that are ischemic |
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Types of hemorrhagic strokes (2) |
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intracerebral (10%), subarachnoid (3%) |
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Factors that lead to hemorrhagic stroke (3) |
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Atherosclerotic plaques Developmental abnormalities (Berry aneurysms, AVMs) |
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Fluid backup >> blood pooling |
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How does valvular disease >> CVA? |
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turbulent blood flow >> thrombus |
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How do arrhythmias >> CVA? |
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blood pooling >> thrombus |
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How does cardiac surgery >> CVA? |
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embolus 2º to jostling sclerotic aa |
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Risk factors for CVA: modifiable (6) |
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HTN, CVD, diabetes, TIAs, hyperlipedemia, smoking |
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Risk factors for CVA: nonmodifiable (5) |
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race, age, gender, heredity, previous CVA |
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sudden severe HA, sudden hemilateral weakness/numbness, sudden loss of vision, unexplained diziness |
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Explain "cascade" of chemical activity leading to damage of cells following CVA |
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cells lacking O2 release glutamate >> glutamate binds to NMDA receptors >> Ca influx into neurons >> activation of Ca-dependent enzymes >> infarction & more glutamate released |
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Most frequent cause of death in acute CVA: |
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Caudal shift of brain tissue 2º edema: |
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Lateral shift of brain tissue 2º edema: |
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Factors determining symptoms of CVA (3) |
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location of damage, size of infarct, presence of collateral circulation |
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Test to differentiate hemorrhagic v. ischemic CVA: |
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ID non-vascular lesion, distinguish ischemic/hemorrhagic stroke |
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anemia (endocarditis, vasculitis), thrombocytopenia, thrombocytosis |
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Serum glucose used to test for: |
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Chest XR used to test for: |
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Meds categories for ischemic CVA (3) |
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anticoagulants, thrombolytic, neuroprotective agents |
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Examples of anticoagulants (4) |
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Heparin, aspirin (antiplatelet), Ticlid (decr platelet aggregation), Plavix (antiplatelet) |
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Time limit for tPA to be effective |
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Examples of neuroprotective agent meds (1) |
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NMDA receptor antagonists |
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Meds categories for hemorrhagic CVA (1) |
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Ca channel blockers (Nimodipine); vasodilator |
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CT, MRI, SPECT/PET, cerebral angiography |
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non-CVA causes, edema, infarction, hemorrhage |
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structural detail, infarct <6hr old |
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SPECT/PET useful for imaging: |
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representation of metabolism |
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Cerebral angiography useful for imaging: |
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CVA Direct Impairments: sensory deficits local loss = ____ damage widespread loss = _____ injury |
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Homonymous hemianopsia 2º to destruction in which aa.? |
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Definition
MCA (optic radiation), PCA (1º visual cortex) |
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Signe Brunnstrom's typical sequence of recovery in hemiplegia (6): |
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Definition
1: flaccid period 2: basic limb synergies; spasticity develops 3: gain voluntary control of synergies; spasticity increases 4: mvmt combos beyond synergies; spasticity declines 5: synergies decline 6: spasticity disappears |
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Spasticity experienced primarily in _______ mm |
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Muscles not normally in UE abnormal synergy (5) |
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lats, teres M, serratus ant, finger ext, ankle inv/DF |
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elevating UE c ext elbow >> ext of fingers |
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resisted abd/add of an LE or UE >> same movement in other |
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Changes in mm motor units 2º to CVA: (2) |
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Definition
decr in # of motor units, abnormal recruitment of motor units |
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Impairments that affect balance, gait, UE fxnl tasks (5): |
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Definition
inefficient mm activation, inability to maintain contraction, incr effort required to produce contraction, incr rxn time, incr mvmt time |
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T/F: Unaffected extremities experience decr force |
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T (~25% decr force in "unaffected" extremities) |
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Hemispheres' roles in motor programming (L v. R) |
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L: involved in sequencing mvmt R: involved in sustaining mvmt ("motor impersistence") |
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Speech/language disorders occur with lesions to ______ hemisphere |
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Visual-perceptual dysfxn usually seen in ____ hemisphere lesions |
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Types of visual-perceptual disorders (2): |
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body scheme disorder, body image disorder |
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Body scheme/image disorders |
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unilat neglect, anosagnosia, pusher syndrome |
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Affective disorder: R hemispheric lesion >> ?? |
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Affective disorder: Depression caused by lesion in ?? |
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Behavioral changes 2º to L hemisphere damage (4): |
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communication problems, difficulty processing info sequentially.
cautious, anxious, disorganized; hesitant to try new things |
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Behavioral changes 2º to R hemisphere damage (4): |
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probs c spatial-perceptual tasks, neglect, difficulty understanding whole idea of task.
quick, impulsive, overestimate abilities; need to get them to slow down |
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rapid onset unilat leg swelling c dependent edema |
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Prevention of skin breakdown (4): |
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positioning, turning schedule, avoid shearing, keep skin dry |
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Posture is all about _____ |
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Postural control is (4 descriptors): |
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automatic & subcortical, dynamic, graded, related to size of BOS |
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REDUCE for low-level pts, INCREASE BOS for fearful/high tone pts |
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Task progression: gravity |
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with gravity >> against gravity |
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Task progression: -metrics |
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small range >> big mid range >> extremes |
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1. trunk control 2. midline orientation 3. head control on trunk 4. limb function on trunk (closed chain >> modified closed >> open chain) |
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Motor Learning Principles used in pt management/treatment (6) |
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1. management v. treatment 2. work within FUNCTIONAL context 3. encourage problem solving 4. part >> whole 5. practice 6. feedback (summary; ea 5-10 trials) |
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Suggested sequencing of treatment (5) |
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establish building blocks of task WB >> NWB iso >> ecc >> conc small arc within midrange >> larger ranges slow >> fast |
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_____________ >> start of UE dysfxn/pain |
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Causes of posterior pelvic tilt in sitting posture (4) |
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Definition
weak trunk ext, weak abd, decr lumbar/thoracic ROM, short hamstrings |
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Levels of muscular control at shoulder (3) |
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Definition
1. scapular stabilizers 2. prime movers 3. R/C |
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Normal scapular alignment (spine, inferior angle, med border/inf angle abd) |
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Definition
spine @ T2-3 inf @ T7 medial border 3-4 fingerbreadths from spine inf angle 4-5 fingerbreadths from spine |
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Causes for scapular malalignment (4) |
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weakness of scapular stabilizers, muscular imbalance of scap stab, trunk malalignment, weight of flaccid arm |
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Sequelae of inf rotation malalignment (5) |
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inf sublux abnormal scapulohumeral rhythm impingement/pain shoulder-hand syndrome (CRPS) decr UE fxn |
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inferior, anterior, superior |
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Inferior subluxation s/sx (3) |
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most common subluxation occurs in flaccid stage not painful (initially) |
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Subluxation reduction (4) |
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correct trunk alignment correct scapular position move humerus (lift, depress, etc.) rotate humerus (ER, IR) |
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Anterior subluxation s/sx (4) |
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occurs in hypertonic stage painful 2º to overactivity of upper traps & levator scap humerus in hyperext/IR |
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Superior subluxation s/sx (3) |
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occurs in hypertonic stage painful voluntary mvmt limited to abd & IR |
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Pusher syndrome: higher occurrence with damage to ___ hemisphere |
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Pusher pts c R-sided lesions show both pushing and _____ |
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Pusher pts c L-sided lesions show both pushing and _____ |
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Area of damage in pushers: |
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posterolateral thalamus (involved in perception of upright body posture) |
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Perception of vertical in stool tilting experiment (Karnath) |
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18º toward side of lesion |
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Pusher syndrome theories (2): |
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1. trying to compensate for mismatch between visual/perceived vertical 2. response to unexpected loss of balance when moving upright |
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Typical components required to diagnose pusher syndrome (3) |
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spontaneous body posture abd/ext of nonparetic extremities resistance to passive correction of tilted posture |
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Pusher syndrome prognosis (2) |
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Definition
pushing usually gone within 6 mos. pts achieve same level of rehab, but 3.6 weeks (63%) longer to do so |
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Transfers with pushers (2) |
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start c low-pivot transfers transfer to less involved side |
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Sequential order of pusher intervention (Karnath) (4) |
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1. realize disturbed perception of upright 2. visually explore surroundings and body's relation to them 3. learn mvmts necessary to reach vertical 4. maintain vertical while performing other activities |
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