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Rooting; Sucking Reflexes |
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Asymmetrical Tonic Neck Reflex (ATNR) |
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Tonic Labyrinthine Reflex (TLR) |
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Symmetrical Tonic Neck Reflex (STNR) |
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Protective Extension Sideways Reaction |
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Protective Extension Backwards Reaction |
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1mos --> brain & spinal cord developed in embryo 8 weeks --> vestibular system starts to form 8-9 weeks -->now a fetus 7-9weeks --> touch sensitivity (mvmt) |
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20 weeks --> auditory system is developed |
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28 weeks --> lung fxn is developed auditory, vestibular, & olfactory systems intact visual system nearly set sensation of light touch |
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3rd week post birth --> end of 1st year |
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Is movement perfected in one skill before another skill is practiced |
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Cephalocaudal Proximal --> distal Asymmetric --> symmetric --> controlled asymmetric Gross motor --> fine motor |
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Sagittal Plane --> Frontal plane --> Transverse plane |
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physiological flexion (extensors elongated), can fixate on objects |
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pelvis is high & in anterior tilt weight on chest & face can lift & turn head enough to clear airway |
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spine is rounded pelvis is perpendicular to surface |
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chest is triangular w/ ribs horizontal thin Intercostal spaces (chest to abdomen ration = 1:3) no respiratory reserve |
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head more laterally oriented LE more extended UE more ER |
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can lift head & turn using hyperextension |
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head & hands are more often in midline |
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active head extension to 90 w/o bobbing UEs in line w/ shoulders |
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more head lifting & pushing up on arms (passive extension) Lateral Flexion- weight of body goes in direction of head turn |
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Can visually follow objs still not developed active extension except in neck |
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holds head up scapula back w/ humeral extension (high guard) |
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More controlled symmetrical mvmt begin to see active extension use rotation in cervical spine w/ head turning & weight shifting |
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hands consistently midline active flexion w/ hands to feet (Rolls) |
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“swimming” “prone prop” begin to see head righting reaction develops into a mature weight shift |
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child has achieved voluntary asymmetry & dissociation trunk muscles=3AG |
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plays w/ feet can bridge rolls to prone |
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mature weight shift on UEs lots of pushing w/ extensors |
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can sit independently in midline w/ good control (Static) |
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occurs w/ support & is static will bounce lack of hip extension |
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chest is now more rectangular but ribs still horizontal |
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worked in 4-pt Belly Crawled weight shift & rotation while sitting |
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active sensory-spatial exploration = visual feedback 4 postural control |
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can sit alone & reach away from midline long sit bear crawl vaulting side sit w/ rotation CREEPING |
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w/ support & still no eccentric lowering |
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Cruising sideways & forwards |
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Independent Movement gained all necessary ROM to develop higher skills mature weight shifts except in standing |
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being upright allowed gravity & abdominals to rotate ribs down 1:1 |
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reflex that appears during gestation or at birth & becomes integrated by about 6mos age
neural circuit is at the spinal cord or brain stem level |
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Postural responses that begin to appear in infancy or childhood as a child develops balance in various positions --> remain throughout life
Righting Rxns / Equilibrium Rxns / Protective Rxns |
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involuntary stereotypic motor response to a specific stimulus |
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body’s accommodation to changes in the center of gravity (orientation in space) reactions seen in the head, trunk, and extremities allows function in upright position with hands free and develops into mature weight shifts |
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Help keep the head oriented in relation to the body adjusts body part to vertical |
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Allow the infant to use the extremities to protect from falls by weight bearing on them Will develop with child in prone, supine, sitting, & standing as the child gains these postures |
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overall state of tension in the body body’s readiness to move & it’s ability to resist downward pull of gravity. |
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resistance of muscle to passive elongation or stretch
flaccid; hypotonia; hypertonia; dystonia |
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increased contractions of muscles causing stiff & awkward movements velocity dependent response to passive stretch presence of a spastic catch “clasp-knife”, clonus, decerebrate & decorticate rigidity |
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resistance to stretch throughout the passive ROM independent of velocity & equal resistance in both directions leadpipe & cogwheel |
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involuntary sustained or intermittent muscle contractions causing twisting/repetitive movements, abnormal postures or both (ACTIVE) -hypertonic vs. hyperkinetic dystonia |
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Cerebellar Impairments to coordination |
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Hypotonia, Dysmetria, Dysdiadochokinesia, Intention Tremor (“titubations”), Dyssynergia & Asynergia, Ataxia, Dysarthria, Asthenia, Rebound Phenomenon, Nystagmus |
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Basal Ganglia Impairments to coordination |
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Bradykinesia & Akinesia, Rigidity, Resting Tremor, Chorea, Athetosis (“wormlike”), Choreoathetosis, Hemiballismus (“flailing”), Dystonia |
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Dorsal Column Impairments to coordination |
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Decreased proprioception, kinesthesia, & discriminative touch |
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Biomechanical/Musculoskeletal
Coordinated normal motor strategies (anticipatory/feedforward & compensatory/feedback)
Sensory Organization (somatosensory; visual; vestibular) |
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Coordinated normal motor strategies |
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Ankle Strategy (small perturbations; distal --> proximal) Weight-Shift Strategy (pelvis, hip ABD & ADD) Hip Strategy (lg perturbations; narrow BOS; COG near LOS; proximal --> distal) -Anterior = abdominals then quads -Posterior = Erector Spinae then hamstrings Stepping Strategy Sitting Strategies & Suspensory Strategy (Protective ext.; lowering COG) |
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-Romberg; Foam & Dome Test; Dynamic Posturography -Functional Reach; Berg Balance Scale; POMA; Timed Get Up & Go; Timed Walking; BES |
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Treatment approach for coordination and balance |
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1)Address vertigo & nausea 1st 2)Safety & fall prevention (environmental factors & lifestyle changes) 3)Address musculoskeletal problems & faulty postural alignement 4)Address Static control 5)Address Dynamic control 6)Address sensory organization 7)Compensatory training if necessary |
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Surgical Managment of amputations |
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remove part of limb allow wound healing construct residual limb |
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Postoperative management of amputation |
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monitor healing prepare limb for prosthesis increase patients strength and endurance provide interim means of performing ADLs |
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foot-ankle assembly shank socket suspension system knee unit |
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1. Nonarticulated Feet (SACH, SAFE or energy-storing/elastic keel) 2. Articulated Feet (Single-axis or Multi-axis=PF, DF, IV, EV) 3. Custom Feet |
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1. Exoskeletal- “Crustacean” -wood or plastic outer shell; body wt carried by walls of shell (heavier) 2. Endoskeletal- “Modular” -central aluminum or rigid plastic pylon covered w/ foam rubber=natural |
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Transtibial - PTB (use with stump sock)
Transfemoral -Quadrilateral (ischial tuberosity & gluteal—narrower ant-posterior) -Ischial Containment Socket (CAT-CAM) (gluteals & sides & bottom of limb—narrower med-lateral) |
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Basic phase of prosthetic training |
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A. Instruction in don/doff prosthesis B. Balance/Pre Gait Exs C. Basic Transitions D. Ambulation in Parallel Bars |
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Intermediate phase of prothetic training |
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ambulation outside of parallel bars (level surfaces) |
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Advanced phase of prosthetic training |
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A. Ascend/Descend stairs B. Step over obstacles C. Stand to floor to back up D. Bending down to get objects |
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Transtibial Prosthetic Gait Analysis Early Stance: |
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1)Excessive Knee Flexion a.Prosthetic Causes: high shoe heel, insufficient PF, socket too far anterior b.Anatomic Causes: flexion contracture, weak quads 2)Insufficient Knee Flexion a.Prosthetic Causes: low shoe heel, excessive PF, socket too far posterior, socket insufficiently flexed b.Anatomic Causes: extensor hyperreflexia, weak quads, anterodistal pn, arthritis |
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Transtibial Prosthetic Gait Analysis Mid Stance: |
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NO ANATOMICAL CAUSES 1)Excessive Lateral Thrust a.Prosthetic Causes: excessive foot inset 2)Excessive Medial Thrust a.Prosthetic Causes: excessive foot outset |
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Transtibial Prosthetic Gait Analysis Late Stance: |
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1)Early Knee Flexion: drop off a.Prosthetic Causes: high shoe heel, insufficient PF b.Anatomic Causes: knee flexion contracture 2)Delayed Knee Flexion: walking uphill a.Prosthetic Causes: low shoe heel, excessive PF b.Anatomic Causes: extensor hyperreflexia |
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Transfemoral Prosthetic Gait Analysis Abduction: prosthetic side in stance |
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a.Prosthetic Causes: long prosthesis, sharp or high medial wall b.Anatomic Causes: ABD contracture, ADD redundancy/roll, laterodistal pain, instability (pt. insecure) |
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Transfemoral Prosthetic Gait Analysis Circumduction: prosthetic side in swing |
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a.Prosthetic Causes: long prosthesis, locked knee unit, loose friction @ knee, inadequate suspension(slips down), socket 2 small, foot PF b.Anatomic Causes: ABD contracture, poor knee control, easier for pt |
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Transfemoral Prosthetic Gait Analysis Trunk Shifts Lateral Bend: prosthetic side in stance |
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a.Prosthetic Causes: short prosthesis, sharp or high medial wall b.Anatomic Causes: ABD contracture, weak ABDs, hip pn, instability |
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Transfemoral Prosthetic Gait Analysis Trunk Shifts: Forward Flexion: prosthetic side in stance |
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a.Prosthetic Causes: unstable knee unit b.Anatomic Causes: instability |
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Transfemoral Prosthetic Gait Analysis Trunk Shifts Lordosis: prosthetic side in stance |
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a.Prosthetic Causes: inadequate socket flexion b.Anatomic Causes: hip flexion contracture, weak hip extensors |
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Transfemoral Prosthetic Gait Analysis Rotations |
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1)Medial/lateral whip: prosthetic side @ heel off (usually probs w/ prosthesis) 2)Foot Rotation @ heel contact (usually probs w/ prosthesis) |
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Transfemoral Prosthetic Gait Analysis Excessive Knee Motion |
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1)High Heel Rise: early swing (flexes too fast & too soon) a.Prosthetic Causes: inadequate knee unit friction, slack extension aid b.Anatomic Causes: forceful hip flexion 2)Terminal Impact: late swing a.Prosthetic Causes: inadequate friction, taut extension aid b.Anatomic Causes: abrupt & forceful hip extension as knee reaches extension |
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Transfemoral Prosthetic Gait Analysis Reduced Knee Motion |
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1)Vault: prosthetic side in swing a.Prosthetic Causes: same as for circumduction b.Anatomic Causes: easier for pt than to control knee unit |
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Level at which the greatest vertebral damage has occurred |
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The most caudal segment of the spinal cord with normal sensory functions as determined by testing the 28 dermatomes on each side of body |
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The most caudal segment of the spinal cord with normal motor function as determined by testing 10 myotomes on each side of the body |
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Neurological level of SCI |
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The most caudal segment of the spinal cord with normal sensory and motor function bilaterally |
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what is the prediction of self-care and ambulation based on in SCI |
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period immediately following the injury during which there is an absence of reflex activity, flaccidity, and loss of sensation below the level of the lesion may last several hours up to several weeks early resolution is good prognostic sign |
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1) Respiratory Impairment* (C1-3 (phrenic nerve) + abdominals, intercostals & pectorals) 2) Spasticity (increase 1st 6 mos, plateau @ 1yr; influenced by many factors) 3) Impaired Temperature Control 4) Autonomic Dysreflexia (Hyperreflexia) = above T6 (HTN, sweating, H/A, spasticity) 5) Postural Hypotension 6) Pressure Sores/Ulcerations 7) Bowel & Bladder Dysfunction (reflex vs. non-reflexic neurogenic B&B) 8) Sexual Dysfunction 9) Pain = traumatic; nerve root; dysesthesias; musculoskeletal pain 10) Miscellaneous: Heterotropic Bone Formation; Contractures; DVT; Osteoporosis; Kidney Stones; other injuries |
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1) Pt & Family Education 2) Respiratory Management (exs: diaphragmatic, glossopharyngeal; strengthening; coughing; support) 3) Maintain or Increase ROM 4) Positioning (prevent skin breakdown & increase tolerance to upright) 5) Selective Strengthening (RESISTANCE is contraindicated the 1st few weeks) |
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Restricted motions for Parapeligia |
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avoid trunk or excessive hip motion (SLR>60; hip & knee flexion >90) |
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Restricted motions for tetraplegia |
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avoid head, neck, & UE motion |
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What is the rate of progression determined by for SCI |
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pt tolerance medical status psychological status certain restrictions due to stabilization procedure |
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What is used as a foundation of PT approach in SCI |
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Mat program
develops strength, ROM, endurance, coordination, & early use of substitutions & compensations for balance & mvmt |
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What are the principles used to increase functional movement in SCI |
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1) Head/Hips Relationship (will inversely affect pelvis & lower trunk) 2) Elbow Locking Mechanism in Tetraplegia (ER & ADDanterior deltoid & pec major) 3) Leverage 4) Force Vectors (direction/line of force) 5) Momentum & Velocity (2+ mvmts must occur for max force) 6) Timing 7) Reverse Action of Muscle Pull (stabilize distally to move proximally) |
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Sequence of learning of activities in SCI |
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develop stability in a position --> develop controlled mobility --> develop fxl skill |
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What are the criteria for ambulation in SCI |
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Adequate strength Adequate ROM Adequate endurance (cardiovascular) Good postural alignment No other medical /health factors that might interfere |
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What is the progression of ambulation with SCI |
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1) Don/doff braces 2) Static & dynamic balance in para-stance in pb 3) Stand to sit in pb 4) Ambulate w/ appropriate gait pattern in pb (swing-to & swing through vs. 4-pt) 5) Sit to stand in pb 6) Ambulate outside of pb w/ crutches 7) Falling & getting up 8) Advanced gait skills |
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what is the ankle locked at for dorsiflexion in an orthosis to have the patient's COG shifted anteriorly |
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5-15 degrees of dorsiflexion |
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What are the types of burns |
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Thermal Electrical Chemical Radiation |
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What are the types of thermal burns |
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-Neck- neutral to slight hyperextension -Shoulder- 90 ABD w/ 10 horiz. ADD w/ slight ER -Elbow- full extension & forearm in neutral -Wrist/Hand- Wrist=0-30EXT, MCPs=50-70FLEX, IPs=straight, -Thumb= btwn ABD & EXT -Hip & Knee- Neutral rotation w/ slight ABD 10-15 & knees EXT -Foot & Ankle- 90 dorsiflexion/neutral, Heel suspended
**Position of comfort = position of contracture** |
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Exercise Contraindications for Burns |
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exposed joints tendon exposure over the PIP jt DVT Compartment Syndrome Fresh Skin Graft |
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exercise precautions for burns |
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heterotrophic bone formation |
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Exercise Target areas for burns |
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areas likely to develop contractures areas over/near jts |
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Active, red, raised, rigid (can still change) |
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Factors affecting scar management |
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prolonged healing time repeated harvesting of donor sites race/increased pigment age location depth tension (across jts) gender (female>male) |
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characterized by limited variation of motor strategies & limited ability to vary motor behavior to address specific situations |
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established risks for atypical development |
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events that have occurred that pose obvious problems i.e: hydrocephalus, chromosomal abnormalities, congenital myopathies… |
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Environmental Risks for atypical development |
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i.e: single parent, maternal age/problems/health |
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